Parcourez les articles et découvrez de nouvelles perspectives!
The case of a woman with tachycardia induced cardiomyopathy due to PJRT detected at the age of 54 years. The patient refused to undergo catheter ablation and was successfully treated with a beta blocker.
We present the diagnostic pathway and management of an asymptomatic patient with a solitary, potentially high-risk coronary plaque, utilizing modern and advanced non-invasive and invasive pathophysiological, morphological, and functional assessment. By using coronary computed tomography angiography, computer-based solutions were employed enabling non-invasive understanding of the plaque characteristics and the local and global functional micro-environment. Invasive coronary angiography-based, computational, and wire-free hemodynamic lesion assessment as well as intracoronary imaging with optical coherence tomography were further utilized, allowing for personalized management and guiding coronary revascularization.
“Professor Meier is nuts!”. This was the main complaint of a 64-year-old patient requesting a consultation. He had been on atorvastatin for two years, prescribed by his primary physician. Because of chest pain he recently underwent coronary angiography performed by Professor Meier. No coronary lesions were found; in fact, since his coronaries were in pristine shape, Professor Meier told the patient that he could stop the statin. “But”, the patient indicated, “my LDL-C (low-density lipoprotein cholesterol) was above 150 mg/dl (3.9 mmol/l) before atorvastatin and now it has been well below 80 mg/dl (2.1 mmol/l). I never missed a dose and have zero side effects. I looked it up and guidelines clearly recommend a statin in cases like me. Also, I have hypertension and as is obvious, am a bit overweight”.